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It is currently believed that short‐term presence of senescent cells can promote tissue repair and attenuate collagen fiber production through secretion of SASP, and also recruit immune cells to accelerate tumor cell clearance, while long‐term accumulation of senescent cells exacerbates chronic inflammation, promotes tissue fibrosis, inhibits tumor local immune cells, and inhibits tumor growth, fibrosis, and tumor local immune cell activity [41]
